Yaya Yayi Bambanci?
Rheumatoid arthritis yana da wahala. Yawancin mutane da cututtuka na rheumatoid za su gaya muku cewa wani lokaci ya wuce kafin an ba su wata ganewar asali. Ko da ma likitan da ake zargi da zaton rutsurancin jini a farkon lokaci, babu wani gwaji ko wani bangare na shaida wanda ya tabbatar da ganewar asali. Sakamakon binciken lafiyar mai haƙuri, tarihin likita, gwaje gwaje-gwajen gwaje-gwajen, da nazarin ilimin lissafi dole ne a yi la'akari.
Ba wai kawai akwai wasu siffofin cututtukan da suka shafi cututtuka na rheumatoid ba, marasa lafiya da ke nunawa da bayyanuwar cutar. A yawancin marasa lafiya na cututtukan ƙwayar cuta, farawa na bayyanar cututtuka yana faruwa a hankali kuma an kafa shi sosai kafin a gane shi. Hanyoyin polyarthritis yawanci sun hada da kananan ɗakunan hannu da ƙafa kafin yin tafiya zuwa manyan haɗin. Amma akwai wasu hanyoyi da maganin arthritis ke nuna kanta wanda ba su da mahimmanci: ƙananan ƙwayoyin cuta (ƙananan ciwo da cike da ciwon da ke ciki, yana shafe ɗaya ko fiye da gidajen abinci, daga bisani ya zama m); Polymyalgic (sau da yawa wani haƙuri mai haƙuri da ƙananan ƙwararru na stiffness, sau da yawa kafadu ko hips); tsarin (wanda ba a nuna ba, kamar zazzabi, malaise , asarar nauyi); Mura ɗaya daga cikin wadanda ke ci gaba da kasancewa (arthritis a cikin wani babban hadin gwiwa da ke ci gaba).
Kwayar wariyar launin fata na farko zai iya bambanta da abin da muke tunanin shi a matsayin maganin arthritis.
Mutane da yawa marasa lafiya suna da alamun kullun na al'ada lokacin da suka fara tuntubi likita game da bayyanar cututtuka. Mutane da yawa suna da nauyin rayuka na yau da kullum. Abin da zai iya zama mafi rikitarwa ga masanin kimiyyar, sabili da haka ga mai haƙuri, shine akwai wasu yanayi dabam-dabam wanda yake da alakar ƙwayoyin maganin polyarthritis wadda ke da alamun da ke da alaka da cututtukan arthritis na rheumatoid.
Saboda irin wadannan yanayi suna kama da cututtuka na rheumatoid, dole ne a yi la'akari da su, sa'an nan kuma a cire, kafin a iya tabbatar da ganewar ƙwayar cututtuka na rheumatoid. Ga wasu daga cikin yanayin da ke nuna cutar arthritis rheumatoid:
Arthritar Post-Gyammancin Bidiyo
Magungunan cututtuka mai cututtuka mai maɗaukaka na iya haifar da polyarthritis da ke kama da ilimin cututtuka na rheumatoid a asibiti. Kwayar cututtuka mai cututtuka, irin su parvovirus B19, yawanci za'a iya bambanta ta tarihi mai rikitarwa, raguwa, da gaskiyar cewa bayyanar cututtuka na ƙarshe na tsawon lokaci. Yana da mahimmanci ga masu bincike su ware ciwon hepatitis na kullum ko cutar kwayar cutar mutum, musamman ma idan ana kula da maganin immunosuppressants.
Matsarar maganin maganin ƙwararraya
Psotic arthritis zai iya zama da wuya a rarrabe daga cututtuka na rheumatoid, musamman ma idan ba ta raguwa ba. Yin haɗin zane-zanen sacroiliac ko haɗin gwal na haɗin hannu zai iya ƙuntata ƙin ganewar asali ga cututtuka na psoriatic. Sauran ƙananan spondyloarthropathies ( cututtuka mai yatsawa , spondylitis , cututtukan zuciya da cututtukan zuciya) suna iya yin maganin rheumatoid arthritis. "Haɗin gwiwa tare da juna, rashin samun haɗin ƙananan haɗi, nau'in alade-kamar alamu, da kuma sanya hannu kan lakabiyar lumbosacral duk suna yarda da maganganun maganganun da suka dace", in ji Cleveland Clinic.
Lupus
Lupus erythematosus na tsarin jiki zai iya hadewa tare da haɗin gwiwa wanda ya haifar da cututtuka na rheumatoid, amma lupus ya bambanta da wuya zama rashin lafiya. Tare da lupus, nakasar zai iya ci gaba da kama da wadanda suke da alaka da cututtuka na rheumatoid - bambancin da yake kasancewa saboda tayin da laxity na ligament tare da lupus, ba halakar haɗin gwiwa ba.
Scleroderma
Kundin jini cikakke, jigidar sunadarai mai zurfi, da kuma nazarin ilimin lissafi (misali, antineclear, antientromere, da antibodisraserase antibodies) ana bada umurni da yawa lokacin da ake zargi da jinya da ciwon scleroderma . Hanyoyin halittar Creatine kinase, nauyin erythrocyte sedimentation, da kuma ma'aunin gina jiki na C-reactive na iya taimakawa yayin da aka samo asali.
"Hanyoyin da aka haifa suna nuna cewa myositis, vascularitis, malignancy, ko farfadowa na sclerosis tare da wata cuta ta jiki," in ji AAFP.
Vasculitis
Rhumatica ( Polymyalgia rheumatica (PMR) da giant cell arteritis (GCA) na iya faruwa tare da polyarthritis symmetrical. Mafi sau da yawa, tarihin likita daga wadannan marasa lafiya zai iya taimakawa wajen rarrabe PMR ko GCA (misali, ciwon kai, tare da kafada da zafi na hip). A wasu lokuta, ganewar asali na iya dogara ne akan kallon cutar nan gaba. A wasu kalmomi, matsaloli masu tsanani za su iya bunkasa a tsawon lokacin da ke nuna zuwa ga vasculitide. Tsarin jiki na jiki zai iya bayyana tare da polyarthritis. Matar granulomatosis na Wegener na iya zama rheumatoid factor tabbatacce.
Osteoarthritis
Babu alamomi da alamun bayyanar cututtuka, farawa a cikin tsofaffi tsofaffi, da kuma tsarin haɗin gwiwa (asymmetric) sau da yawa ne don gano bambancin osteoarthritis daga arthritis rheumatoid. " Magungunan osteoarthritis na iya samun mummunan bayyanar jarrabawa, amma yana da ma'anar sabanin PIP da farko, ba tare da haɗin synovitis ba, ba RF (kwayoyin rheumatoid) ba ne, kuma yana da tasirin radiyo," in ji rahoton Cleveland Clinic.
Gout
Gida mai yawan gaske yana haɗuwa da ƙwayar ƙwayar cuta guda ɗaya ko ƙananan ciwon jini da ciwon zuciya, na tsawon kwanaki 3 zuwa 10. Amma, hare-haren hare-haren zai iya zama mafi sauƙi, na ƙarshe, kuma ba zai warware ba, wanda zai haifar da aikin arthropathy. Ayyukan arthropathy na iya haifar da lalata da haɗin gwiwa. An bambanta shi daga cututtukan arthritis na rheumatoid saboda rashin rabon sararin samaniya da ke ragewa da kuma rashin ciwon osteopenia , kamar yadda Cleveland Clinic ta ce.
Tsaro
CPPD crystal shaida cuta ne nasaba da wani tsararru na na asibiti manifestations. Zai iya zama mai tsauri, tsutsa (kamar yadda ake amfani da shi), rudun maganin maganin ƙwaƙwalwar ƙwayar cuta, ko kuma osteoarthritis-like. An rarraba tsinkayyar ta hanyar manyan hare-haren synovitis wanda ke nuna gout. Ana amfani da nazarin ruwa mai gina jiki don ganewar asali.
Sjogren ta ciwo
Sjogren ta ciwo shi ne muni, cututtukan ƙwayar cuta. Zai iya faruwa a matsayin yanayin farko ba tare da wani cututtukan rheumatic ko a matsayin yanayin na biyu ba tare da wani yanayin rheumatic. Ƙungiyar Sjogren na farko zai iya yin maganin rheumatoid arthritis. Kimanin kashi 60 zuwa 70 bisa dari na marasa lafiya na Sjogren na da kyau ga matsalar rheumatoid. Har ila yau, akwai ciwo, ƙwaƙwalwa, da kuma kyawawan ƙarancin ɗakunan. Tsara ta musamman na iya taimakawa wajen rarrabe tsakanin ciwo da Sjogren da rheumatoid arthritis.
Sarcoidosis
Sarcoidosis iya bayyana tare da synovitis a cikin mahalli da yawa kuma zai iya zama rheumatoid factor tabbatacce. Amma, wasu halaye na taimakawa wajen rarrabe shi daga rheumatoid arthritis. A wasu lokuta, za a iya buƙatar da kwayar cutar nama don kafa ganewar asali.
Fibromyalgia
Fibromyalgia za a iya kasancewa da nuna bambancin launin fuska da tsayayyar zuciya, amma rashin synovitis, rashin ciwo a kan motsi, da kuma ɗakunan binciken al'ada da kuma nazarin hotunan nazarin ilimin fibromyalgia, ya bambanta shi daga cututtukan arthritis, a cewar Cleveland Clinic.
Layin Ƙasa
Akwai yanayin da ke nuna cutar arthritis, musamman a farkon wannan cuta. Babban halayen na yau da kullum shi ne polyarthritis. Tarihin likita na likita, kallon bayyanar cututtuka da cututtuka a cikin lokaci, da kuma ƙarin gwajin gwaji ya taimaka wajen gane yanayin da ake samu daga cututtuka na rheumatoid.
Sources:
Gout da Tsaro. Feyrouz Al-Ashkar. Cleveland Clinic.
http://www.clevelandclinicmeded.com/medicalpubs/diseasemanagement/rheumatology/gout-and-pseudogout/
Rheumatoid Arthritis. William S. Wilke. Cleveland Clinic.
http://www.clevelandclinicmeded.com/medicalpubs/diseasemanagement/rheumatology/rheumatoid-arthritis/
Sclerosis / Scleroderma: Tsarin Magungunan Hanyoyi. Monique Hinchcliff, MD da John Varga, likitan likitancin Amirka. Oktoba 15, 2008.
http://www.aafp.org/afp/2008/1015/p961.html
Binciken asalin Arthritis na Rheumatoid: Abin da Mai Bukata ba ya Bukatar Sanin. E Suresh, MD MRCP. Jaridar Royal Society of Medicine. Satumba 2004.
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1079582/