Maganganun Cikin Gida Za su iya nuna alamar Ci gaba na Ciwo na HIV
Hairy leukoplakia (wanda aka fi sani da laukoplakia mai gashi, ko OHL) shine labaran da ake gani a cikin mutanen da ba a yarda da su ba, wadanda suke nunawa da fararen launi a gefen harshe da halayyar "gashi". Yana daya daga cikin cututtuka da dama wanda ke iya magance matsalolin mutane da yawa, a mafi yawan lokuta idan adadin CD4 na mutum ya fadi a kasa 200 kwayoyin / mL.
OHL ne ke haifar da cutar Epstein Barr (EBV), kwayar cutar iyali wadda ta shafi kusan kashi 95 cikin 100 na yawan jama'a, kodayake mafi yawan zasu bunkasa rigakafin da zai hana cutar daga bayyanawa. Duk da haka, a cikin mutanen da ke dauke da kwayar cutar HIV, aikin kulawa da rashin lafiyar da ke hade da kamuwa da cuta ya ba OHL damar samun nasara. Saboda haka, an dauke shi da kamuwa da cututtuka ta hanyar HIV (OI) .
Rashin raunuka na OHL sunyi mummunan kuma basu sa wani bayyanar cututtuka. Maimakon haka, yanayin shine alamar maƙasudin kare lafiyayyen mutum da kuma ƙarawa mai karuwa ga wasu, mafi tsanani. Kullum magana, OHL ya bayyana sau da yawa a cikin maza fiye da mata, kuma yana da wuya a gani a cikin yara.
Kafin zuwan maganin rigakafi (ART) , OHL ya kasance mai girma game da cigaba da cutar zuwa karshen lokaci, inda yawanci 47% suka ci gaba da cutar HIV zuwa AIDS a cikin shekaru biyu.
A yau, tare da farkon da aka fara samar da ART, yanayin da OHL ya haifar da shi ya fi muhimmanci, ba kamar sauran cututtukan da ke dauke da kwayar cutar HIV ba, waɗanda suka fi ƙarfin gaske da kuma kasancewa a matsakaicin matakan CD4.
A halin yanzu, bincike na baya-bayan nan ya nuna cewa shan taba , a haɗe da ƙananan CD4, yana haifar da kusan yawan sau biyu a haɗarin OHL.
Bincike, Jiyya da Rigakafin OHL
Harkokin OHL sun bambanta da girman. Zaka iya gabatarwa a kowane ɗaya ko bangarorin biyu na harshe ko a cikin kunci. Ba su da yawanci ciwo ba sai dai akwai sakandare na biyu, kamuwa da cuta.
A wasu lokuta, raunuka za su iya bayyana fili, yana sa ya fi wuya a bambanta da wasu, irin wannan cututtuka. Duk da haka, ba kamar labaran da za a yi ba, ba za a iya cire HSL daga harshen ba. Hakanan, tare da launi sunaye, sune halaye mafi ban sha'awa na OHL a cikin mutanen da ke dauke da kwayar cutar HIV.
Yayin da dubawa na asibiti sau da yawa isa ya goyi bayan ganewar asali, wasu bincike sun nuna cewa kashi 17 cikin dari na nazarin gani ba daidai bane. Idan ana buƙata, za a iya gane ganewar asali tare da nazarin binciken kwayoyin halitta da sauran hanyoyin bincike don tabbatar da cutar EBV.
Saboda ciwon OHL ba shi da kyau, babu magani da ake bukata. Duk da haka, a wasu-musamman wadanda ke fama da girman CD4 na Zovirax (acyclovir) na iya taimaka wajen magance yanayin. Duk da haka, hawan OHL zai sake tashi sau ɗaya idan an dakatar da farfadowa acyclovir sai dai idan aikin rigakafi yana mayar da hankali.
Hakan zai iya komawa lokacin da cutar ta HIV ta bunkasa kuma masu shan magani na marasa lafiya sun zama marasa tasiri a kan maganin cutar HIV.
Yin rigakafi na OHL yana dogara ne akan samin asali da maganin cutar HIV, tare da jagorancin Amurka na yau da kullum suna bada shawarar farawa ART a CD4 ƙidaya tsakanin 350 da 500 kwayoyin / mL, har ma a CD4 ya fi sama da 500 Kwayoyin / mL.
An ƙaddamar da katse shan taba don hana bayyanar OHL, da kuma sauran masu cutar HIV da kuma marasa lafiya na HIV.
Sources:
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Cherry-Peppers, G .; Daniels, C. Meeks, V .; et al. "Gabatarwa a cikin zamanin HAART." . Jaridar Ƙungiyar lafiya ta kasa. Fabrairu 2003: 95 (Karin bayani 2): 21S-32S.
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