Lokacin da batun cutar cutar sankarar ya tashi, mutane da yawa suna da fahimtar cewa wannan zai iya zama cutar yara. Amma bayan haka, cikakkun bayanai suna neman samun ɗan damuwa. Ya kamata a lura da cewa, yayin da cutar sankarar ita ce mafi yawan ƙananan yara , shi ma gaskiya ne cewa ƙwayar cutar sankarar ƙanƙara ba ta da kyau.
Kwanin vs vs m cutar sankarar bargo
Laukemias mai zurfi suna cigaba da sauri.
Kwayoyin da ake kira ƙananan ƙwayoyin cuta-sune balaga ba ne kuma ba su ci gaba da isa ba don aiwatar da ayyukan su na rigakafi. Sabanin haka, ciwon leukemias na ci gaba ne a cikin ƙwayoyin da suka bambanta ko kuma balagagge, wanda zai iya yin wasu ayyuka, amma ba sosai. Kwayoyin mahaukaci na leukemias na kullum sukan ninka a hankali kadan fiye da leukemias mai tsanani. Duk da haka, yawancin leukemias suna da matukar wuya a yara .
Yawancin leukemias yara sune cutar kututtukan lymphocytic mai tsanani (ALL). Yawancin lokuta da suka rage su ne m cutar cutar sankarar myeloid ( AML ). Ma'anar 'myeloid' da 'lymphocytic' a cikin waɗannan sunaye sunaye zuwa gidaje biyu daban-daban na sel: wadanda zasu haifar da jinin jini na lymphocyte (lymphocytic); da kuma wadanda za su haifar da jinin jini, tallan, da sauran kwayoyin jini mai kama da monocytes, neutrophils, da sauransu.
Kwayar yara vs. Ciwon sankarar cutar ta jiki
Duk da yake cutar sankarar bargo ne mafi yawancin malignancy a ƙuruciya, cutar cutar sankarar jini ta kasance mai wuya, wanda ya ƙunshi kasa da kashi 1 cikin dari na leukemias yara.
An bayyana yanayin a matsayin cutar sankarar bargo da ke faruwa a cikin makonni 4 zuwa 6. Ana tunanin cewa wannan mummunan fara farawa kafin a haifi jariri.
Abin takaici, tsawon watanni 6 na rayuwa shine kashi ɗaya bisa uku, duk da tsananin ilimin chemotherapy. Duk da haka dai an ba da tabbacin maganganun ba da sanarwa ba, amma kimiyya ba ta ba da amsoshin tambayoyin yadda wannan zai faru ba.
Gaskiyar cewa wadannan mu'ujjizai masu ban al'ajabi sun ruwaito, zasu iya aiwatar da tsarin yanke shawara na likita don likitoci da iyaye. Wasu sunyi mamaki ko mahimmancin tsarin kulawa, musamman a farkon, yana da tabbacin.
Ya kamata a lura da cewa ilimin leukemias ba shi ne kawai za a bayar da rahoto ba. Sauran cututtuka na yau da kullum na faruwa a wasu magungunan cutar sankarar bargo, kuma remission na iya zama mai saurin rai, ko gajeren lokaci, ko mafi tsayuwa, dangane da rahoton da aka ruwaito.
Karamin cutar sankarar ƙanƙara na yara
Ko da yake cutar sankarar bargo ne mafi yawan ciwon daji a yara da matasa, dukkansu, ƙwayar cutar sankarar yara ya kasance cuta mai wuya. Kimanin kashi 75 cikin 100 na leukemias tsakanin yara da matasa sune ALL, kuma mafi yawan sauran lokuta shine AML.
Bisa ga nazarin kididdigar da Cibiyar Kankara ta Amirka ta yi, DUKU ya fi kowa a lokacin yaro, yana tsakanin shekaru 2 zuwa 4. Sakamakon lokuta na AML sun fi yaduwa a fadin ƙananan yara, sai dai AML ta zama ɗan lokaci fiye da shekaru 2 da kuma lokacin shekarun ƙuruciyar.
Dangane da kabilanci da kabilanci, DUKU ya fi yawa a tsakanin 'yan asalin Safan sa da yara masu fari fiye da' yan Afrika da Amirkawa da na Asiya, kuma yana da yawa a cikin yara maza fiye da 'yan mata.
AML yana faruwa a daidai tsakanin yara maza da 'yan mata na dukkan jinsi.
Leukemias na yau da kullum suna da wuya a cikin yara, amma idan sun faru, yawancin wadannan sun kasance marasa cutar sankarar jini (CML), wanda ke kula da kara yawan matasa a cikin matasa fiye da kananan yara. Kwayar cutar sankarar ƙananan yara (JMML) ta kasance a cikin yara ƙanana, tare da shekaru kimanin shekaru 2.
> Sources:
> Fozza C, Bellizzi S, Bonfigli S, Furofesa, Duro F. Longinotti M. Cytogenetic da Harkokin Halitta na Harkokin Halitta a cikin Kwayar cutar Kankarar bargo. Eur. J. Haematol. 2004; 73: 219-222.
> Jain N, Hubbard J, Vega F, Vidal G, Garcia-Manero G. Borthakur G. Tsayar da cutar ta Kanjamau ta Myeloid: Labarin Magunguna uku da Nazarin wallafe-wallafe. Clinic cutar sankarar bargo. 2008; 2: 64-67.
> Martelli MP, Gionfriddo I, Mezzasoma F, Milano F, Pierangeli S, Mulas F. Arsenic Trioxide da All-Trans Retinoic Acid Target NPM1 Mutant Level Oncoprotein da kwance Apoptosis a NPM1-Muted AML Cells. Jinin jini . 2015; 125: 3455-3465.
> Verhaak RGW, Güdswaard CS, van Putten W, Bijl MA, et al. Yankewa a cikin Nucleophosmin (NPM1) a Magungunan cutar sankarar myeloid mai zurfi (AML): Ƙungiyar tare da Sauran Abubuwa na Halitta da Tsarin Halittar Halitta da Gabatarwa Ta Ƙaddamar Halitta Harshen Sigina da Suhimmancin Ƙwararrun Masarrafar. Jinin jini . 2005; 106: 3747-3754.
> Wiemels J. Harkokin Watsa Labarai game da Abubuwan Cutar Lurarre. Harkokin hulda na Chemicobiological . 2012; 196 (3): 10.1016 / j.cbi.2012.01.007.